TREATMENT RECOMMENDATIONS FOR AIDS
(from http://www.virusmyth.com)
AIDS need not be treated by drugs alone. In fact, the drugs themselves appear to be the cause of the disease in many cases.
Treatment recommendations based on the work of Dr. Heinrich Kremer, Hamburg, Prof. Alfred Hassig, Berne, and Dr. Stefan Lanka, Stuttgart.
The many and varied diseases that define the AIDS syndrome: fungal infections of the lung, of the mucous membranes, the brain, and the gut, and
the degenerative changes in the endothelial cells of blood vessels and lymphatic vessels (KS), occur because of an ongoing heightened production of
gaseous nitricoxide and oxygen radicals in immune cells and other cells.
Under these conditions CD4 helper cells mature predominantly to Th-2 cells, which migrate to the bone marrow, where they activate defences against
bacteria by producing antibodies, but only few to Th-1 cells mesurable in plasma, which attack fungus and virus infected cells. If this situation persists, a higher quantity of proteins of the cytoskeleton and of
mitochondria is released as en effect of heightened cell decay. Against these proteins a higher rate of the antibodies are formed, which are detected by the HIV-antibody tests. Once a certain, arbitrary level is
reached, the patient is declared "HIV positive".
A persistently elevated level of nitricoxide and oxygen radicals comes about as a result of:
Ongoing contact with antigens (e.g. from infections, injuries or operations)
Inhalation of nitrites ("poppers") which are stored in cells as NO2, and which on increased exposure to calcium ions (e.g. through physical exertion) are released, which affects the endothelial cells of blood vessels and lymphatic vessels with a small capillary diameter, and leads thereby to degenerative changes (swollen lymph nodes and finally to Kaposi Sarcoma).
Impairment of the mitochondria, the single cell energy suppliers, which synthesize the energy-carrying-molecule ATP, which is used for all functions of the organism
The causes of chronic mitochondrial damage are:
Damage of its DNA due to antibiotics (e.g. sulpha compounds such as Septrime, TMPSMX), which block the synthetisation of folic acid and purine,
and exhaustion of its thiol-pool by these antibiotics, by additives in vaccines (ex. hepatitis-b) and by cytostatica like AZT, ddI and ddC, which bind the SH-groups of glutathione and cysteine.
Reduced glutathione produced resulting from liver damage, e.g. chronic hepatitis (occuring in hemophiliacs and intravenous drug consumers),
excessive alcohol consumption, or through shortage of nutritional cysteine, esp. in developing countries.
Glutathion molecules reduce oxygen- and nitricoxydemolecules, so that ATP production in mitochondria is not disturbed. An ongoing shortage of glutathione means that Th-1 cells poison themselves attacking fungi and virus containing cells.
Reduced oxygen transport in cells because of oxidation (methhaemoglobinaemia) which exceeds the reductive capacity of glutathione. This comes about because of the strongly oxidising effect of nitrites (poppers), antibiotics and nucleoside analogues (AZT and others).
Lack of plant antioxidants which bind to toxic degradation products (oxygen radicals) and thereby reduce inflammation and stressreactions.
On prolonged impairment of mitochondria, they dissolve their symbiosis with the host ("Warburg Phenomenon"). Cells then increasingly switch over to producing energy by anaerobic fermentation, which results in excess lactic acid production, and the growth of fungi and opportunists, and ultimately to wasting, at which point cells obtain essential nutrients directly from the myoproteine. By an heightened activity of reverse transcription the cell
nucleus then saves its genotype.
By means of:
A supply of sulphur compounds in sea salt, mineral water and algal products, e.g. agar, of cysteine and methionine containing protein mixtures, e.g.
N-acetyl-cysteine, curd and whey can stimulate glutathione formation in cells especially in the liver. Glutathione may also be administered intravenously.
Plant antioxidants, which bind to toxic oxygen decay products, and natural protease inhibitors, e.g. heparine and heparinoids in klamati- and kelp algae, which activate the body's own anti-proteases and bind to cations that attack the cell walls, can slow down chronic inflammatory reactions with increased cell division.
Co-enzyme Q10 and NADH and high doses of Vitamin C and E can improve electron transport in the respiratory chain of cells. Folic acid, thiols and
low doses of selenium, (e.g. brewers' yeast), and zinc can support the repair of damage to mitochondrial DNA.
Essential fatty acids in linseed [flax] oil, thistle oil, hemp oil, soya oil mixed with curd, which enhighten the uptake of oxygen in cells.
Herbs like Carduus Marianus (Milk Thistle) can restore liver function, and partly fermented lactic acid beverages can restore gut flora.
Ethereal oils, rubbed on to the chest and in the armpits serve to stimulate the immune system through the ground substance (matrix)
Extract of grape fruit kernels (Citricidal) and gargling with honey/vinegar are helpful against fungal infection.
Targeted stress reduction techniques, e.g. autogenic training, stretching and massages, and refraining from excesive physical exercise by using
perfomance-enhancing drugs, e.g. coffee, alcohol, nicotine, amphetamines (X-tasy), cocaine, heroin and poppers.
Avoiding inflammatory reactions and overexertion, and considering, without fear, safer sex rules.
Of a nourishment poor in sugar but rich in fiber, with much high-value carbohydrates, plant antioxidants, e.g. vegetables, fruit, herbal and green teas, cold-pressed oils, partly fermented dairy products, [fermented] soya beans, and fish, but not iron-rich red meat.
...a flexible resistance in people with AIDS and HIV positives can be restored.
Progress achieved by these measures to bolster the immune system can be monitored by measuring stress hormone profiles, the T4/T8 cell ratio,
macrophage activation (neopterine test) and cutaneous anergy, the glutathione level in plasma and in T-4 helper cells.
HIV, which is held to be responsible for causing 30 different AIDS-defining diseases, has never been shown to be transmissible nor self-reproducing; it
has never been isolated, photographed or otherwise properly characterised, as required by the established rules of virology. The original experimental technique of Gallo and Montagnier in 1984, involved culturing cells from AIDS patients with leucaemic cells and embryonal cells, activated with
hydrocortison, that show a high activity of reverse transcription. This effect of an artificially amplified reverse transcription was then interpreted as signifying the presence a new retrovirus. A virus-specific enzyme could not be prooved according to the established rules.
Synthetic protease inhibitors, which are supposed to inhibit the formation of essential viral building blocks, over time, cause malaise, diabetes, kidney stones and liver failure in patients given them. After PIs and
nucleoside analogues are first given, an apparent short-lived decline in inflammatory reactions and "virus production" may be observed, but it then
rises again, which is attributed to resistance developing. Just as on long-term administration of antibiotics, nucleoside analogues, which are
only ever 1% incorporated into the cell nucleus, cause, by damaging mitochondria, serious damage to the brain, to the bone marrow, muscles and
internal organs.
Study group for AIDS therapy
c/o Felix A. de Fries
Eglistrasse 7
8004 Zurich (Switzerland)
Tel/fax (0041 1) 401 3424
For a related article, see "15 years of Aids"
http://www.virusmyth.com/aids/data/ah15years.htm
Though I disagree with Hassig's recommendations for soybean and hemp oils and his suggestion that oral thrush can be treated with a vinegar and honey mixture (tea tree oil would be more appropriate), this treatment protocol for AIDS is one worth pursuing.
From Stephen Byrnes, ND, RNCP
http://www.PowerHealth.net
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